Lab Members


Rebecca M. Lynch, Ph.D.
Assistant Professor

Rebecca M. Lynch, Ph.D., joined the GW Department of Microbiology, Immunology and Tropical Medicine University in February 2016 as an Assistant Professor.

Dr. Lynch graduated with a B.S. in Molecular, Cellular and Developmental Biology from Yale University in 2003. She worked for two years as a research technician before embarking on a PhD in Immunology and Molecular Pathogenesis 
at Emory University under supervision of Dr. Cynthia Derdeyn. During her graduate studies, Dr. Lynch characterized the role of the HIV envelope in the escape from neutralizing antibodies during HIV subtype C infection, and received two Young Investigator awards and three NIH/NIAID scholarships. Following her graduation in 2010, Dr. Lynch performed postdoctoral studies investigating the properties and origins of broadly neutralizing HIV antibodies at the Vaccine Research Center directed by Dr. John Mascola at the National Institutes of Health (NIH). Dr. Lynch has published over 20 research articles to date and is the holder of a K22 career transition award.


Maria KoromMaria Korom, M.D.
Senior Research Associate and Lab Manager

Maria received her medical degree from the the Semmelweis University Medical School in Budapest, Hungary where she was a pediatrician for 8 years before moving to St. Louis, Missouri. She conducted her postdoctoral studies in Saint Louis University where she conducted in research on Drosophila Nipped-B protein, a model for human Cornelia de Lange syndrome. In her second postdoctoral position, she studied proteins involved in Herpes Simplex-2 (HSV-2) virulence and virus escape as well as ways to improve HSV-2 vaccine efficiency. She described the first four polypeptides produced from the gamma34.5 open reading frame of HSV-2. During this time, she also received her Executive MBA with a focus on International Business from the John Cook School of Business in St. Louis. Maria joined the Lynch lab in April of 2016 and is focused on studying replication defects in HIV-1 resulting from virus escape from broadly neutralizing antibodies.